Oxygen toxicity
نویسندگان
چکیده
منابع مشابه
Oxygen Toxicity
Prolonged exposure to oxygen at high pressure can have toxic effects, particularly on the central nervous system,but at pressures used clinically it does not pose a problem. The main topics discussed hereare:
متن کاملOxygen Toxicity.
Oxygen, the gas vital to sustain life, can also destroy it. It may become toxic at an elevated partial pressure, which may be the result of a rise in inspired oxygen concentration, an increase in environmental pressure or a combination of both. The toxicity tends to express itself most recognizably in one of several forms including central nervous system manifestations, pulmonary signs and symp...
متن کاملCNS oxygen toxicity.
This short manuscript will briefly present our current knowledge about CNS oxygen toxicity: the clinical manifestations, descriptions and incidence of symptoms, and the time – duration relationship that defines the safety limits and risk factors leading to enhanced toxicity to hyperbaric oxygen. A very general outline will present suggested mechanisms underlying hyperoxia-induced seizures and s...
متن کاملCO2 exacerbates oxygen toxicity
Reactive oxygen species (ROS) are harmful because they can oxidize biological macromolecules. We show here that atmospheric CO(2) (concentration range studied: 40-1,000 p.p.m.) increases death rates due to H(2)O(2) stress in Escherichia coli in a dose-specific manner. This effect is correlated with an increase in H(2)O(2)-induced mutagenesis and, as shown by 8-oxo-guanine determinations in cell...
متن کاملOxygen toxicity: an introduction.
Although oxygen has been known to be toxic for more than 200 years, the clinical importance of oxygen toxicity was not appreciated until an epidemic of retrolental fibroplasia occurred in the early 1950s. Oxygen at high partial pressures is toxic to the respiratory, cardiovascular, nervous, and gastrointestinal systems. Toxicity results from the formation of oxygen-free radicals. These arise wi...
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ژورنال
عنوان ژورنال: Anaesthesia
سال: 1978
ISSN: 0003-2409,1365-2044
DOI: 10.1111/j.1365-2044.1978.tb08373.x